By Gisèle Pickering, Stephen Gibson
This ebook specializes in the reciprocal interactions among discomfort, emotion and cognition. overseas specialists document on their findings, scientific event and the latest literature at the complicated family members among those 3 domain names. scientific, mental, behavioral and neuroimaging techniques converge in the direction of supporting sufferers being affected by ache, cognitive impairment or emotional distress.
Beyond the pathophysiological points of soreness, problems encountered with sufferers are analyzed and methods are proposed to optimize remedy, doctor-patient relationships, and sufferer well-being.
This paintings will entice a large readership, from scientific clinical practitioners to psychologists, nurses and ache specialists.
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Additional info for Pain, Emotion and Cognition: A Complex Nexus
2013). This polysynaptic heterosegmental reflex, which is characterized by a flexion of the stimulated limb, occurs in a time window (approximately 90–180 ms) consistent with the conduction velocity of A∂ nociceptive afferents (Sandrini et al. 2005). Moreover, the threshold of the reflex also coincides with pain perception thresholds, and the amplitude of the reflex increases with perceived pain, suggesting that modulation of NFR amplitude by emotions may reflect spinal nociceptive processes (Sandrini et al.
3 Cerebral and Spinal Modulation of Pain by Emotions and Attention 41 The fact that negative emotions can produce either hyper- or hypoalgesia also raises the possibility that both effects can sometimes compete with one another. For instance, al’Absi and Petersen (2003) observed that public speaking also induced increases in self-reported levels of distress that predicted increases in pain ratings independently from SBP-mediated analgesia. The resulting net analgesic effects of their public speaking task therefore appeared to have resulted from a competition between SBP-mediated hypoalgesia and negative mood-induced hyperalgesia.
An expression style referring to attempts to suppress feelings of anger) contribute to a large extent in explaining the impact of perceived injustice on pain intensity and may also be one of the mechanisms through which perceived injustice influences depressive symptoms (Scott et al. 2013). Attribution of blame is part of the process, as an antecedent of perceived injustice and anger reactions. , to traumatic events) contributes to greater levels of anger and higher perceived injustice, eventually increasing feelings of hopelessness and helplessness and leading to the more or less conscious adoption of a victim role.